If it was really working so well on rGBM, why did almost half of those who progressed opt out of continuing with DCVax?
Liau claiimed 20 years after understanding psPD that it messed up her P3 trial. So why should we believe she finally figured it out with the rGBM in this trial? I'm sure most were mis-labeled psPD and thus the "great" results were also mis-labeled.
Up to about 85% of all rGBM tumors are mesenchymal phenotype. This transition creates the opportunity for L to work even better at transition for certain patients although the best overall opportunity for effect begins with treatment at first diagnosis of GBM or, as we are finding out from other trials, perhaps even at the lower grade levels of brain tumor diagnosis. Best wishes.