AI
Saturday, August 20, 2016 5:18:46 PM
Gabor ..a simple answer to your simple question
I do not know if the mean LDL level in R-IT will be higher or lower then the mean LDL level in I-IT and I don't think it matters since I think I-IT is largely irrelevant for predicting what might happen in R-IT .
JELIS 2ndry is more relevant but here I think you and the other Uber bulls are extrapolating JELIS 2ndry results on questionable assumptions .
Optimal dose high intensity Statins will lower the event rates in any at risk CV event population studied...in both arms. Its the remaining residual risk we are concerned with ...either high TG's and / or low EPA.
I don't see a further 30-40% reduction in events once both arms are optimized on high intensity Statins .
You do .
Since I'm currently long AMRN , I'll be quite happy if you are right ...in proving me wrong ..
Kiwi
I do not know if the mean LDL level in R-IT will be higher or lower then the mean LDL level in I-IT and I don't think it matters since I think I-IT is largely irrelevant for predicting what might happen in R-IT .
JELIS 2ndry is more relevant but here I think you and the other Uber bulls are extrapolating JELIS 2ndry results on questionable assumptions .
Optimal dose high intensity Statins will lower the event rates in any at risk CV event population studied...in both arms. Its the remaining residual risk we are concerned with ...either high TG's and / or low EPA.
I don't see a further 30-40% reduction in events once both arms are optimized on high intensity Statins .
You do .
Since I'm currently long AMRN , I'll be quite happy if you are right ...in proving me wrong ..
Kiwi
