News Focus
News Focus
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BonelessCat

12/09/09 12:25 PM

#25821 RE: mkendra #25819

>>"And the fact that the treatment can be considered only a temporary cure, because after treatment, it is possible to be re-infected with the virus,"

In nearly all viral infections, the virus compromises the immune system in some way, whether it is by overwhelming the system with viral matter (Ebola) or hijacking key elements for its own use (HIV). Seymour has already talked about how following infection and nanoviricide treatment, it appears that immune systems are educated against the virus and can more rapidly respond to new infections or reinfections.

Second, there are no approved treatments for viral infections, other than neuraminidase inhibitors, which also does not include the immune system; those instead inhibit either entry or exit of viral matter infecting host cells. The only things are vaccines, which actions are prophylactic and have no effect after infection. Vaccines are not treatments.
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cabel

12/09/09 12:29 PM

#25822 RE: mkendra #25819

I thought I read somewhere that ANTIBODIES are produced in the body,.. even while our CIDE is killing the virus.
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petekoz1

12/09/09 12:38 PM

#25823 RE: mkendra #25819

Below are comments from Dr. Seymour on this subject, from my post of July 27, 2009. I believe this hypothesis is being tested as part of FluCide (pan-influenza) studies currently underway at Thevac, LSU.

“The scenario you suggest isn't really a vaccine. That's pre-exposure prophylaxis (PEP). A vaccine stimulates the immune system to produce an antibody IN CASE you're infected at a later date.

What you're suggesting has interested me for a long time. For example, let's say that the first case of swine flu hits a community in California. You immediately treat those initial cases (the index cases) as well as the still healthy cluster cases (those in contact with the index cases.) That way you prevent the geometric spread of the disease.

Now if in the meantime you WERE infected but FluCide knocks out the infection quickly, the trigger to stimulate the immune response was the first few hours of circulating virus. Essentially, that's a "live attenuated virus vaccine trigger" so you now have destroyed the virus with the drug and also have the immune system stimulated to produce antibodies...a double whammy in that FluCide knocks out the virus immediately but you have long term protection against that particular strain. I call it a "live attenuated virus vaccine trigger" because even though the virus may be lethal, we nail it so quickly that it can't do any damage.

This hypothesis has to be tested but it appears that it should work.

I also think that we can engineer FluCide to remain active in the body for an extended period of time so it could be used as prophylaxis in health care workers.”


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Ubertino

12/09/09 1:32 PM

#25824 RE: mkendra #25819

Are we NOW in the FDA testing process? SRI and Thevac are testing and as they are familiar with the FDA requirements will their testing for NNVC be acceptable to the FDA? I don't see why not, just so long as the FDA is agreeable prior to the testing and/or after the fact.
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Ovidius

12/10/09 6:57 AM

#25829 RE: mkendra #25819

this might all be true about the FDA steps, but the pumpers here have been predicting huge price gains for in the short term, infact we are a few years past most of those prediction.