FT825 should work in EBV related cancers
Both EBV and HPV manipulate chemokine profiles to enhance viral survival:
EBV-infected cells tend to increase pro-inflammatory chemokines, potentially leading to immune cell recruitment but also immune exhaustion.
HPV-infected cells often suppress chemokine responses to avoid detection, with selective increases in chemokines like IL-8 in tumor stages to create a supportive environment for cancer progression.
This alteration of chemokine profiles helps both viruses persist and, in some cases, contributes to the development of virus-associated cancers.
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