“Brilacidin actually KILLS the virus so viral load HAS to drop dramatically, and the SOC is currently near 0 (a total waste) so how could efficacy not be pronounced? “
Very easily. Just one of the ways it could fail is if its in vitro MoA is due to phospholipidosis (which has nothing to do with claims that that is ann vivo side effect as others have misunderstood).
Consider also that remdesivir is more effective than brilacidin in vitro but only marginally effective in vivo.