Kevetrin induced a stable monoubiquitinated mutant p53, which form accumulates and retains ability to induce apoptosis. This seems quite different than "degrades mutant p53," wouldn't you agree?
They have to pick something. Kevetrin may:
(1) create a stable form of mutant p53 which accumulates and still works (vs. unmodified mutant form which presumably doesn't work) [I would argue that this induction of a stable form that still works is equivalent to restoration of mutant p53 function, but I may be wrong].
or
(2) degrade or remove mutant p53
Can it really do both - stabilize and degrade? Yes, confusion reigns.
But on a day like today, who really cares?! We have a billion dollar antibiotic and all time high, even if nobody really knows how Kevetrin may work !!
Market Data 
Markets 
AI
