I truly apologize that I am in some way confusing you - but in that post I was clearly comparing influenza 'cross-breeding' to hcv 'cross-breeding' only as an illustration of what I meant by 'cross-breeding' at the macro level. For the purposes of this thread I couldn't care less whether hcv 'cross breeds' via monogomous sex, loves deviance or has giant orgies - only that both viruses (flu and hcv) do perform mix-and-match-between-strains RNA recombo.
Again, I would suggest you have somehow confused my intent. The whole thread (and every one of my subsequent posts) started with me proposing an evolutionary mechanism for hcv that involved RNA recombo - I have stayed on the same topic.
PS I found the data you posted interesting as an explanation about why some viruses are better able to do recombination than others, but mostly not pertinent. Not trying to insult your knowledge - just not hugely pertinent to the debate I started and wanted to play out.
Hmmm, suggest you re-read my post. I was clearly referring to evolutionary pressure in the abstract or in the context of pressure on the HCV genome - and there is no question that, for instance, the NS5A inhibitor is a 'threat to the existance' of the hcv within that environment ('environment' = a single homo sapiens).
But agree that you might do well to ignore me since I believe we tend to speak at different levels - and it just seems to cause you angst.
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