I draw a distinction between: a) ritonavir boosting to convert a BID/TID drug into a qD drug; and b) ritonavir boosting to try to salvage a drug that has a small (or nonexistent) therapeutic window.
What ITMN is doing with ITMN-191 falls within case b, and that’s why I’m skeptical of the outcome.
“The efficient-market hypothesis may be the foremost piece of B.S. ever promulgated in any area of human knowledge!”