why would genotype 2/3s still be treated 48 weeks once their is a direct anti-viral on the market? Unless you are referring to the fact that the G2/3 trials are lagging?
Geno-2/3 patients have been harder to cure than geno-1/4 patients for any drug regimen I know of that’s been tried against all of the HCV genotypes. Moreover, among the three main classes of direct antivirals, only the nukes offer pan-genotypic coverage; by and large, the non-nukes and protease inhibitors in development today have been tailored for genotype 1.
“The efficient-market hypothesis may be the foremost piece of B.S. ever promulgated in any area of human knowledge!”