virosomes we used do not contain phosphatidylserine, so the interaction of 24 KIM-1 with this phospholipid cannot explain the KIM-1 mediated facilitation of uptake
. Our 3 finding that TW-37 blocked entry of the SARS-CoV-2 virosomes suggests prevention of viral internalization 4 as an approach to anti-SARS-CoV-2 viral treatment. This would be reminiscent of the use of Enfuvirtide, 5 Maraviroc and Ibalizumab for HIV where administration of agents that will bind to the virus in the circulation 6 will compete for tissue binding (Henrich and Kuritzkes, 2013).