I'm not a financial advisor all my posts are based on my own personal unique way to do DD, nothing very technical or professional.
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Very thin, takes someone to sold 40 shares for a bit of copper to took it down. Let's hope this doesn't repeat again other than a mistake
After the 11M wall at 8's There's just the 50M wall at 11's, they know what's going on.
In the last PR they just spoke about an upcoming adquisition, there should be another PR for the progress and for shareholder value, which wasn't the subject of the last PR. That'll give shareholders more confidence on what's about to come.
Why not? I've seen some companies put that info on their fillings, it'd be great if they do, and in more detail.
Right Curnc, filings are coming this week and we might see about the plans they have. I believe this short week will see some action and disclosure, has been holding strong on that 7-8 levels for over a week, regardless of the BTC drop, since last time they posted in Twitter by june, BTC had a significant growth, no convertible notes, makes this stock very attractive
What the public might be asking is " how this stock would benefit from having cryptocurrency and not cash in hand?". Well One Bitcoin generated value is at least $45,000 in cash, and will be increasing, when you make Bitcoins, you can sell not only One Whole Unit but parts of it and get cash to whatever you want to. And when you can make a Bitcoin, there's not limits on how much cash you can make as long as those machines are working.
The question would be. Where we at? Like, there's fees involving BTC generation, they had been generating BTC since 2021, and.giving some dividends last year, at this point obviously there should be more BTC created, plus they are about to adquiere more mining machines which means more BTC generated. And with increasing BTC value, that means they gonna have revenues to make this stock grow in shareholder value, not only thru dividends but thru buybacks or share cancellations. All needed is revenues, and is not that you are trying to sell stuff to get revenues, you have ( in other words) a machine to make money. All In My Opinion
When sells from the BID is dumping, but I've seen lots of ASK walls fall, which is a good sign. Those selling at the BID/ASK 7/8, are paper hands, those selling at 0.0006 gotta be Shorters closing positions
There's manipulation of course, and this clown called "skyrocket" is living proof, paid basher. Why is he so determined to lie and spread confusion among this board? Though the Share Structure has been updated daily, this clown still says that there's dilution going on? How? There's no way possible if OTC markets Transfer Agent update that info everyday. Why he wants the price to drop so bad??? He's working for someone ( Shorters)
Take a look a the " Trades" #64 thru #67, the BID/ ASK was 0.0007 - 0.0008 this guy was able to sell almost 4M at 0.0006, what da heck. Shorters closing positions at 0.0006??? Very suspicious... Bid wall of 126M, for me Shorters trying to close IMO. Let's see some moves tomorrow after this approval. Saw lots of 0.0007 walls falling as well.
Clown 🤡🤡🤡 alert, skyrocketinsight
All due respect dude, who the heck ask you? You are persuading people to sell, to buy into your scam? Move on man, move on. The CEO is about to give news this coming week. Not only financials increase shareholder value ( just the news about it, very unlikely ever an OTC has increase it's value from revenues, that's on NASDAQ), but share cancellation, buy backs, mergers, purchases, etc. You never know, you are clearly a paid basher.
How many accounts do you have?
Ask at $0.0008 Got destroyed... beautiful
I don't think the sec had approved anything yet. They still saying that Wednesday should be the approval date.
Tbh, after some DD, the most important resistance wall that fell was by 6.6M at 0.0008, at 11:22am. which make the SP dip, and so other people did so ( sold )I believe they flipped, they went from Vtxb to cgac, all IMO, they were pumping cgac today so I believe some flippers sold and bought into cgac, but I saw some recovery on vtxb at EOD. I believe vtxb is way [img][/img]more solid structurally and something is cooking there, I'm pretty sure if not this week, or next, but sooner or later the fate of vtxb is to go high, potentially to pennies. I don't see vortex stuck on trips, very unlikely, this is for longs IMO and specially seeing BTC growing strong.
At the same time I noticed an important group of people buying 14M shares at the ASK at 0.0007 instead of panic sell at 0.0006, they took advantage of this dip. Interesting, let's see what happens this week...
BTC is hitting new highs since December 2021, right now at $47,000, let's hope it hits 50K soon...that's the consequence of this approval...as of RN.
A PR would put the cherry on the top
Right man, this days are crucial for the future of this company, sec approval should be a catalyst and hopefully we hear some communications from the CEO. Silence and bad news make the SP fall since people sell and move on to the next, communications assure holders that the CEO is working on something and is not planning to get stuck at trips IMO
Hopefully, last time he posted on Twitter was back in July, to announce dividends, but putting the company current last December speaks for itself, that means something should be simmering.
I just called them and left a message:
213-260-0321
Email: info@vortexbrands.us
Spooz has 5.4B OS five more millions and will max out the AS number and had run 1000%. When people look at VTXB it shouldn't stop til pennies
You're going down
For me is very clear that surfbear is a paid basher, he is all over the place trying to stop all runners to persuade people to sell, that way shorters can open a position and make profits when SP falls. All this guys are getting investigated and will be indicted for their illegal practices.
There's a way to find out, all pink current companies has the obligation to report everything they do, that way we don't have to speculate and we actually can see it, and decide if we like it or not, invest or withdraw.
Why this guy keeps denying that there's an ongoing manipulative short sells going on at OTC stocks??...is not the first time I hear this criminals denying their own criminal activities...
( From the OTC short report site:)
About Us
OtcShortReport.com was put together for one reason. To track the true volume, and display in a coherent fashion the daily short positions of over the counter stocks. Short Positions for over the counter (OTC) stocks are hard to come by. At OtcShortReport.com we make digesting the information easy to understand, and add valuable charting and historical data. This saves you time, and most importantly it is all free!
We hope to help investors with understanding what is really happening with your OTC investments.
Believe it or not, most CEO's are not even aware that they are a victim of manipulative short selling!
We always look forward to comments and suggestions. If there are any features you would like to see, or anything we can help with making your experience a better one, please don't hesitate to contact us.
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CLINICAL TRIALS & FUNDING
Not long ago our good friend TT posted all the achievements of TSI without fundings. Around 2.5B shares were issued in about 10 years. I know of companies that just issued 2.8B shares in 3 months and are trading at $0.001, unlike TSI, which speaks of the great capabilities of our CEO Tim Dixon, Thomas, James and the BOD.
Let's see what 2.5B shares did for TSOI, this is without fundings...
65 filed patents
20 published patents
3 issued patents
(5) INDs w #’s
JadiCell/CTE Excl license
Treated (5) Nsvy SEALS successfully/CTE via RTT
International Amazon Prime sales
StemVacs Platform
Campbell Neurosciences
Allogen Biologics
CampbellCell
Campbell Score
ApoptoCyte
NanoStilbene
QuadraMune
Projuvenol
NanoCannabidiol
NanoPlus
NanoPSA
DermalStilbene
Now having $10M available, besides the ongoing Phase III clinical trial, do you know how many other potential clinical trials are waiting in line? And for a company to get a shot for a Clinical Trial, the inventors gotta be really really good at it, let's take a look of what could go thru clinical trials and how effective they are...
THERAPEUTIC MONOCYTES FOR PREVENTION OF SUICIDAL IDEATION
Type: Application
Filed: March 4, 2022
Publication date: September 8, 2022
Applicant: Therapeutic Solutions International, Inc.
Inventors: Thomas E. Ichim, Timothy G. Dixon, Famela Ramos, Kalina O'Connor, James Veltmeyer
Suppression of Inflammation Induced Memory Dysfunction by Cord Blood Derived Monocytes
The lipopolysaccharide administration model was used to replicate neuroinflammation associated with suicidal behavior. Female BALB/c mice were treated with control, daily lipopolysaccharide treatment (10 ng/mouse0, and some with cord blood derived monocytes. Cord blood derived monocytes were extracted by ficoll separation of cord blood mononuclear cells and 4-hour incubation on T175 plastic flasks. Non-adherent cells were washed off by rinsing with phosphate buffered saline and adherent cells where subsequently dissociated using trypsin and placed into another flask. Cord blood monocytes where cultured for 24 hours with 1 IU of oxytocin per million cells in complete DMEM media with 10% fetal calf serum. Cells were dissociated with trypsin, washed in phosphate buffered saline and administered intravenously vial tail vein at a concentration of 500,000 per mouse on days 0, 3, and 6.
To assess memory function, water filled basin which was 120 cm in diameter was broken into 4 quadrants. 10 cm diameter platform placed 1 cm below water. Mice were forced to swim to find the hidden platform, starting from all four different quadrants, each day for a total of 7 days. As seen below, mice receiving umbilical cord monocytes resisted the pathological effects of lipopolysaccharide treatment.
Suppression of LPS Induced Plasma Interleukin-6 by Cord Blood Derived Monocytes
The lipopolysaccharide administration model was used to replicate neuroinflammation associated with suicidal behavior. Female BALB/c mice were treated with control, daily lipopolysaccharide treatment (10 ng/mouse0, and some with cord blood derived monocytes. Cord blood derived monocytes were extracted by ficoll separation of cord blood mononuclear cells and 4-hour incubation on T175 plastic flasks. Non-adherent cells were washed off by rinsing with phosphate buffered saline and adherent cells where subsequently dissociated using trypsin and placed into another flask. Cord blood monocytes where cultured for 24 hours with 1 IU of oxytocin per million cells in complete DMEM media with 10% fetal calf serum. Cells were dissociated with trypsin, washed in phosphate buffered saline and administered intravenously vial tail vein at a concentration of 500,000 per mouse on days 0, 3, and 6.
To assess interleukin 6 levels in plasma, mice were sacrificed at the indicated timepoints and cytokine was assayed by ELISA.
EX VIVO GENERATION OF IMMUNOCYTES RECOGNIZING BROTHER OF THE REGULATOR OF IMPRINTED SITES (BORIS) EXPRESSING CANCER STEM CELLS.
Type: Application
Filed: February 22, 2022
Publication date: August 25, 2022
Applicant: Therapeutic Solutions International, Inc.
Inventors: Thomas E. Ichim, Timothy G. Dixon, Feng LIN, Famela Ramos, James Veltmeyer
EXAMPLES
Stimulation of Immunity to CD133 Positive PC-3 Cancer Stem Cells
Cord blood generated allogeneic dendritic cells were obtained by culture of adherent monocytes with GM-CSF (100 IU/ml) and IL-4 (100 IU/ml) for 7 days. Cells expressed CD11c and possessed typical dendritic morphology. Cells where incubated with full length BORIS protein and treated with 30 ng/ml Poly (IC) for 24 hours to induce dendritic cell maturation. Dendritic cells where cultured with allogeneic peripheral blood mononuclear cells for 7 days in the presence of IL-2 (10 IU/ml) and anti-CD3 and anti-CD28 beads (10(5) beads per ml. T cells where purified by Magnetic Activated Cell Sorting for CD3 (containing both CD8 and CD4 cells). PC-3 prostate cancer cells where cultured and separated into either control (no T cells added), unfractionated, CD133 negative and CD133 positive. Cells where co-cultured at a 1:1 ratio and cytotoxicity was determined by MTT assay.
As observed, T cells possessed a preferential ability to kill CD133+ cells, which corresponds to a cancer stem cell phenotype. Results are shown in FIG. 1.
Stimulation of Immunity to Fik Positive PC-3 Cancer Stem Cells
Cord blood generated allogeneic dendritic cells were obtained by culture of adherent monocytes with GM-CSF (100 IU/ml) and IL-4 (100 IU/ml) for 7 days. Cells expressed CD11c and possessed typical dendritic morphology. Cells where incubated with full length BORIS protein and treated with 30 ng/ml Poly (IC) for 24 hours to induce dendritic cell maturation. Dendritic cells where cultured with allogeneic peripheral blood mononuclear cells for 7 days in the presence of IL-2 (10 IU/ml) and anti-CD3 and anti-CD28 beads (10(5) beads per ml. T cells where purified by Magnetic Activated Cell Sorting for CD3 (containing both CD8 and CD4 cells). PC-3 prostate cancer cells where cultured and separated into either control (no T cells added), unfractionated, flk negative and flk positive. Cells where co-cultured at a 1:1 ratio and cytotoxicity was determined by MTT assay.
As observed, T cells possessed a preferential ability to kill PC-3 flk+ cells, which corresponds to a cancer stem cell phenotype. Results are shown in FIG. 2.
Stimulation of Immunity to Rhodamine Positive PC-3 Cancer Stem Cells
Cord blood generated allogeneic dendritic cells were obtained by culture of adherent monocytes with GM-CSF (100 IU/ml) and IL-4 (100 IU/ml) for 7 days. Cells expressed CD11c and possessed typical dendritic morphology. Cells where incubated with full length BORIS protein and treated with 30 ng/ml Poly (IC) for 24 hours to induce dendritic cell maturation. Dendritic cells where cultured with allogeneic peripheral blood mononuclear cells for 7 days in the presence of IL-2 (10 IU/ml) and anti-CD3 and anti-CD28 beads (10(5) beads per ml. T cells where purified by Magnetic Activated Cell Sorting for CD3 (containing both CD8 and CD4 cells). PC-3 prostate cancer cells where cultured and separated into either control (no T cells added), unfractionated, Rhodamin negative and Rhodamin positive. Cells where co-cultured at a 1:1 ratio and cytotoxicity was determined by MTT assay.
As observed, T cells possessed a preferential ability to kill PC-3 Rhodamin+ cells, which corresponds to a cancer stem cell phenotype.
STIMULATION OF NATURAL KILL CELL MEMORY BY ADMINISTRATION OF DENDRITIC CELLS
Type: Application
Filed: February 8, 2022
Publication date: August 11, 2022
Applicant: Therapeutic Solutions International, Inc.
Inventors: Thomas E. ICHIM, Timothy G. DIXON, James VELTMEYER, Famela RAMOS
Example 1 Homotaurine Increases Ability of StemVacs to Stimulate NK Activity
Peripheral blood mononuclear cells where obtained by ficoll centrifugation and plated with StemVacs umbilical cord derived dendritic cells. StemVacs was generated by culture of umbilical blood adherent cells with interleukin 4 and GM-CSF for 7 days with maturation step induced by 24 hour culture TLR agonist. Assessment of natural killer cell activity was performed subsequent to culture of cells with taurine (10 micrograms/ml) or homotaurine (10 micrograms/ml) for the indicated timepoints. Cytotoxicity against NK target cell line was performed using the flow cytometry Promega assay. Results are shown in FIG. 1.
Example 2 Homotaurine Increases Ability of StemVacs to Stimulate T Cell Activity
Peripheral blood mononuclear cells where obtained by ficoll centrifugation and plated with StemVacs umbilical cord derived dendritic cells. StemVacs was generated by culture of umbilical blood adherent cells with interleukin 4 and GM-CSF for 7 days with maturation step induced by 24 hour culture TLR agonist. Assessment of T cell activity was performed subsequent to culture of cells with taurine (10 micrograms/ml) or homotaurine (10 micrograms/ml) for the indicated timepoints. T cell activity was assessed by ELISA quantification of interferon gamma production after stimulation with 5 micrograms per ml of phytohemagglutinin.
IMMUNOTHERAPY FOR OPIOID ADDICTION
Type: Application
Filed: December 20, 2021
Publication date: June 23, 2022
Applicant: Therapeutic Solutions International, Inc.
Inventors: Thomas Ichim, Timothy G. Dixon, Famela Ramos, Wais Kaihani, James Veltmeyer, Kalina O'Connor
Example 1: Reduction of Brain Microglial Activation by PRP and Pterostilbene
BALB/c mice were anesthetized with isofluorane and administered pterstilbene (0.4 mg/mouse) and/or platelet rich plasma (5 microliter per mouse) via intranasal route subsequent to induction of systemic inflammation by intraperitoneal administration of endotoxin. Mice were sacrificed after 24 hours of treatment and quantification of cytokine IL-18 was performed by ELISA from brain homogenate tissue. As seen below, a synergistic reduction of TNF-alpha was observed. Results are shown in FIG. 1.
Example 2: Reduction of Brain Microglial Activation by PRP and Oxytocin
BALB/c mice were anesthetized with isofluorane and administered oxytocin (0.1 IU/mouse) and/or platelet rich plasma (5 microliter per mouse) via intranasal route subsequent to induction of systemic inflammation by intraperitoneal administration of endotoxin. Mice were sacrificed after 24 hours of treatment and quantification of cytokine IL-18 was performed by ELISA from brain homogenate tissue. As seen below, a synergistic reduction of TNF-alpha was observed. Results are shown in FIG. 2.
TREATMENT OF MAJOR DEPRESSIVE DISORDER AND SUICIDAL IDEATIONS THROUGH STIMULATION OF HIPPOCAMPAL NEUROGENESIS UTILIZING PLANT-BASED APPROACHES
Type: Application
Filed: December 8, 2021
Publication date: June 9, 2022
Applicant: THERAPEUTIC SOLUTIONS INTERNATIONAL, INC.
Inventors: Thomas Ichim, Timothy G. Dixon, James Veltmeyer, Kalina O'Connor
Example
BALB/c mice where exposed to random stress 4 times a day by spinning by the tail for 15 seconds. Controls where not stressed. Stressed mice where treated with Prozac or QuadraMune™ daily. Assessment of endogenous neurogenesis was performed by administering BRDU and assessment of incorporation by histology. Augmented neurogenesis was seen in animals which received Prozac, with enhanced neurogenesis in animals taking QuadraMune™
PROTECTION AND REGENERATION OF NEUROLOGICAL FUNCTION BY USING STEM CELLS
Type: Application
Filed: October 27, 2021
Publication date: April 28, 2022
Applicant: Therapeutic Solutions International, Inc.
Inventors: Thomas E. Ichim, Timothy G. Dixon, Amit N. Patel, Famela Ramos, Kalina O'Connor
Examples
JadiCell Conditioned Media Inhibits Endothelial Death.
Injury associated with inflammation. Inflammation causes oxidative stress. Oxidative stress induces death of endothelial cells. Endothelial death exposes basement membrane, induces coagulopathy. Culture of HUVEC cells with upstream inflammatory agent TNF-alpha or downstream H2O2 results in death. Conditioned media (CM) from JadiCells decreases endothelial cell death. Results are shown in FIGS. 1 and 2.
JadiCell™ Reduces Immunogenicity of Endothelial Cells
Endothelial cells can act as antigen presenting cells. Stimulation of T cells by endothelial cells results in inflammation and breaking of blood brain barrier. HLA expression on HUVEC was utilized to quantify one aspects of endothelial antigen presentation. Mixed lymphocyte reaction used as a test of T cell activation. Results are shown in FIGS. 3 and 4.
JadiCell™ Reduces Thrombogenicity of Activated Endothelial Cells
In response to inflammation endothelial cells induce clotting by stimulation of extrinsic coagulation pathway. Tissue factor is activator of extrinsic pathway. HUVEC cells were treated with endotoxin to stimulate activation. Assessment of Tissue Factor performed by flow cytometry. Results are shown in FIG. 5.
CELLULAR, ORGAN, AND WHOLE-BODY REJUVENATION UTILIZING CORD BLOOD PLASMA AND PTEROSTILBENE
Type: Application
Filed: November 4, 2020
Publication date: May 6, 2021
Applicant: Therapeutic Solutions International, Inc.
Inventors: Thomas E. Ichim, Timothy G. Dixon
EXAMPLE
Cord Blood Plasma Suppression of Senescence Marker Beta Galactosidase is Augmented by Pterostilbene
Foreskin fibroblasts where obtained from ATCC and cultured according to manufacture instructions. Accelerated senescence was induced by exposure to indicated concentrations of H2O2 for 48 hours. Cells where cultured in control media (DMEM) or 10% cord blood plasma, or combination of cord blood plasma with 3 uMol Pterostilbene. Senescence was detected by fixing cells with 4% paraformaldehyde and SA-ß-Gal was stained using senescent cells histochemical staining kit (Sigma Aldrich, St. Louis, Mo., USA). Three images per each well were collected, and the SA-ß-Gal-stained cells were counted.
THIS IS JUST TO SHOW A FEW OF THE EXPERIMENTS AND RESULTS SEEN ON THE EXAMPLES, THOUGH TSOI ALREADY HAD SUCCESSFUL TRIALS IN THE PAST, WITH THIS REPUTATION,.I HAVE NO DOUBTS THAT TSOI IS GOING TO SHAKE THE WHOLE BIOPHARMA INDUSTRY.
EDUCATE YOURSELF
Let me explain how an experiment work.
COMMON SENSE
Every Biopharma since very long long time ago, after they medicated a human with certain medicine and that human died, many years ago, the Entity in charge of the pharmaceutical approvals decided to use rats for experiments? Why? Because the life of a human being is consider more valuable.
LETS START!!!
So how i know a medicine works in a rat???
Thru induction ( this is just one of several methods). When you induce some kind of chemicals into an animal which will cause the condition you are targeting, then the game is on.
Once you prove that the rat is infected, then you can try your method to see if it does works or not. For example, let see some extract from the TSOI US Patent for Nutraceuticals for suppressing indolamine 2,3 deoxygenase Patent (Patent # 11,229,674)
Type: Grant
Filed: Oct 23, 2020
Date of Patent: Jan 25, 2022
Assignee: Therapeutic Solutions International, Inc. (Oceanside, CA)
Inventors: Thomas E. Ichim (Oceanside, CA), Timothy G. Dixon (Oceanside, CA), James Veltmeyer (Oceanside, CA)
Primary Examiner: Aaron J Kosar
Application Number: 16/901,028
EXAMPLES ( this is how the experiment was conducted)
Example 1: QUADRAMUNE™ Preserves Memory in Inflammation Associated Memory Decline Model
Female BALB/c mice were treated with control, daily LPS treatment, and some with LPS and QUADRAMUNE™.QUADRAMUNE™ was administered daily by gavage at a concentration 1 (100 ug of broccoli sprout extract, Nigella sativa, and green tea extract, and 50 ug of pterostilbene), and concentration 2 (200 ug of broccoli sprout extract, Nigella sativa, and green tea extract, and 100 ug of pterostilbene).
To assess memory function, water filled basin which was 120 cm in diameter was broken into 4 quadrants. 10 cm diameter platform placed 1 cm below water. Mice were forced to swim to find the hidden platform, starting from all four different quadrants, each day for 7 days. The time was recorded if they could find the hidden platform in 60 s . . . . If not, mice are guided toward the platform and allowed to stand on it for 10 s. Results are shown in FIG. 1.
Example 2: QUADRAMUNE™ Reduces Inflammation Associated Kynurenine Elevation
Blood samples were collected from mice in Example 1 and assessed for Kynurenine content. Augmented levels of kynurenine were observed in LPS treated mice as compared to controls. Results are shown in FIG. 2.
QUADRAMUNE HAD BEEN SCIENTIFICALLY PROVEN TO HAVE GREAT RESULTS. THIS ARE THE CLAIMS
:
Claims
1. A method of inhibiting indolamine 2,3 deoxygenase (IDO) expression or activity, comprising: administration to a patient in need a therapeutic combination comprising: a) Green Tea and/or extract thereof; b) Blueberry and/or extract thereof; c) Nigella sativa and/or extract thereof; and d) broccoli and/or extract thereof.
2. The method of claim 1, wherein said green tea extract is epigallocatechin-3-gallate or an analogue thereof.
3. The method of claim 1, wherein said blueberry extract is pterostilbene or an analogue thereof.
4. The method of claim 1, wherein said Nigella sativa extract is thymoquinone or an analogue thereof.
5. The method of claim 1, wherein said broccoli extract is sulforaphane or an analogue thereof.
6. The method of claim 1, wherein inhibition of IDO expression and/or activity in the host is associated with enhancement of natural killer cell activity.
7. The method of claim 6, wherein said natural killer cell activity is quantified by ability to lyse a virally infected cell.
8. The method of claim 6, wherein said natural killer cell activity is quantified by ability to lyse K562 cells.
9. The method of claim 6, wherein said natural killer cell activity is quantified by ability to lyse YAC-1 cells.
10. The method of claim 1, wherein inhibition of IDO expression and/or activity in the host is associated with enhancement of interferon production.
11. The method of claim 1, wherein inhibition of IDO expression and/or activity in the host is accomplished by enhancement of T cell activation.
12. The method of claim 11, wherein said T cell activation is induction of T helper cell 1 activity.
13. The method of claim 12, wherein said T helper cell 1 activity comprises production of interferon gamma.
14. The method of claim 11, wherein said T cell activation is induction of T cytotoxic cell activity.
15. The method of claim 1, wherein said patient is suffering from a tumor, and said therapeutic combination is administered at a dosage and frequency sufficient to suppress growth of a tumor.
16. The method of claim 15, wherein said tumor growth in the host is associated with suppression of cancer angiogenesis.
17. The method of claim 1, wherein said patient is suffering from COVID 19.
18. The method of claim 1, wherein the patient is suffering from memory loss.
19. The method of claim 1, wherein the patient has elevated levels of Kynurenine
AND I INVITE YOU TO THE JUSTIA SITE TO FIND HOW THE EXPERIMENTS WERE CONDUCTED SUCCESSFULLY FOR EVERY PATENT.
DO YOU SEE HOW TSOI IS WORKING TO IMPROVE THE LIFE QUALITY OF MILLIONS OF PEOPLE THAT COULD BE OUR RELATIVES?
Please do your own DD.
Of course that makes sense. When you buy on the ASK you are filled in one second, and people buy on the ask when chasing a stock. But that's price action, another subject.
So let's take a look at today's chart:
Trade #11, #12, #13 total 70000 shares, BID FILLED AT $0.0221 ASK at $0.0234 ( Gotta be real dumb to buy at the asked price unless you like to give your money for free).
That's 3 purchases. Why appears as sells?
And that's in ALL OF THEM.
And that's for ALL BUYS. Amaizing.
Ok so, let's see, if today I feel like buying 1 million shares, and the price is at like mmm... $0.0229 let's say, I place my BUY order at $0.0228 right, and in the Level II you will see my order at the BID at 1'000,000 at 0.0228, then the price hits 0.0228, and my BID order gets filled, and i adquiere 1'000,000 shares today.
And so others do the same process.
That means that WE ARE BUYING OR WE ARE SELLING? So according to your logic if i buy I'm actually selling?
Explain me the sense of that.
What are you talking about???
Market Capital of 2/08/22 : $54,105,505
Stock price of 2/08/22 : $0.0234
54,105,505÷0.0234= 2'312,201,068 shares
The OS still the same, what are you talking about?????
Those are expertise market. They are able to buy when others can't. For example when price gets at a place when no one can buy or sell they do. Let's say the SP is 0.0002 and 0.0003 all day long goes back and forth, and all of a sudden the SP gets to 0.00025, where no one can either buy or sell, THIS BROKERS CAN. Other than that they buy and sell as regular traders would, usually very tiny orders, sometimes to send "MM signals" to tell other MMs which direction the price should go, but that's a whole other deal, i think we can't go by those " signals" since anyone can buy an order with those amounts and confuse traders.
I'm glad you asked. Wish the Ihub "Trades" chart still showed yesterday's transactions one at a time to show you,.but you will see it today that If you PLACE A "BUY ORDER" IT'LL APPEAR AS A SELL. I'LL EXPLAIN IT TO YOU ONCE WE START TRADING TODAY, LET'S GO TO THE "TRADES"CHART AND YOU WILL SEE THAT EVERY " SELL" IS A " FILLED BID" WHICH MEANS PEOPLE IS BUYING RATHER THAN SELLING.
WHEN WE TRADE THERE'S AN " ACTUAL PRICE", THERE'S A BID ( people trying to buy at lower price), and an ASK (people trying to sell at a higher price obviously
), when the BID is filled is someone purchasing, when the ASK is filled someone is selling. In that chart looks the opposite. I'll show you in 3 hours, I'm glad all transactions are numbered, to make it easier to show you.
Actually almost 4:1 Many loading up
You mean buying volume 3:1
Understanding Short Selling and 2021 P2
Short selling could be risky in a bullish market since you might get 1000 shares if the price goes up then you gonna have to pay those shares back to a higher price. Ask shorters at GME last year, they had to paid hundreds of millions of dollars.
Manipulation of the market comes when this guys put " walls" to make sure the price stays at certain bounds. When you see big amount of shares on the ASK is not always people trying to sell their positions, but is market manipulators trying to stop the price from going higher, that way they can safely keep short selling while everybody safely closed their positions and earn money.
So it's not the Companies fault that price goes up and down, but those guys who has all the money, hedge funds and a dark pool to make millions of dollars by short selling, since the SEC established bounds to limit it, this guys use the dark pool to play by their own rules and keep making money.
But IMO since the Market had been hurt, it's not on shorters interest that the market stays bearish forever, since they won't be able to make money shorting anymore.
So it should get bullish anytime IMO
Understanding Short Selling and 2021 P1
When Biden took office hundreds of companies in the Energy Sector " resurrected" after being in a coma for years at $0.0001 i witnessed ( i said " I" because if i say " we" someone might say " i didn't"), i witnessed an incredible increased not often seen in the Stock Market. From October to April,market was real Bullish, and when the Bullish Spree stopped the Shorters kidnapped the market.
GME had a $18 SP back then and shorters were using it to short sell everyday since there wasn't much of price action, and was " safe" to trade, so a group of young man at Reddit decided to massively buy stock at GME which took the price from $18 to around $320, which was one of the biggest short squeeze In trading history, and put hundreds of shorters in trouble.
Taking advantage of a bullish market this guys did it, but then Shorters took revenge. They started shorting the whole market and affected everybody.
Short Selling is a kind of trading that is the opposite as we usually trade. For example I opted short selling and I get 1000 shares at $1. That's $1000. If the price falls to $0.80, now I earned $200.
You might say! No you actually lose $200. No sir, I didn't purchase by bidding as a regular trade, I opted for Short Selling.
Now imagine not 1000 shares but 1M how would that be?
Some basics on nasdaq and OTC PART 3
Given that situation, we have come to the conclusion that OTC current companies have to use shares and debt to be able to research and develop, pay audits, filings, salaries, etc., what's the purpose?
To be able to create products, technologies, etc , that generate the cash flow needed to sustain the company, so that way the company can be debt free, stay current, increase Adquisition, productions, expand, etc., which generates interest in the Trading Community and Investors, that see the great potential of the company, that way OTC companies increase in SP and increase their value.
Why I trust TSOI? Because in my opinion this company is working very hard to research and development and is going real far with new PATENTED discoveries which potentially will create a loooot of interest in the market community, and will help millions with uncurable illnesses finding a cure and a solution to their medical needs.
This is just my personal opinion.