Thursday, May 12, 2016 11:58:04 PM
It seems that Adam17 with direct links to exposure of flipped PS...and iRhoms 1 and 2 are essential upstream regulators of ADAM17-dependent EGFR signaling
http://www.pubfacts.com/author/Thorsten+Maretzky
https://www.researchgate.net/publication/276462016_iRhoms_1_and_2_are_essential_upstream_regulators_of_ADAM17-dependent_EGFR_signaling
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Now we know many oncologists require / advise testing for EGFR mutation and I'd say PS Targeting is closer than we think if news continues
http://www.questdiagnostics.com/testcenter/testguide.action%3Fdc%3DTS_LungCancerMutation_Panel
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Recommendation
In the provisional clinical opinion, ASCO recommends that if a doctor is thinking about giving an EGFR TKI to a patient with advanced non-small cell lung cancer who has not previously had chemotherapy or an EGFR TKI, the doctor should test the tumor EGFR mutations to determine the most appropriate treatment. Testing for EGFR is done on the sample of tumor removed during a biopsy.
http://www.cancer.net/research-and-advocacy/asco-care-and-treatment-recommendations-patients/epidermal-growth-factor-receptor-egfr-testing-advanced-non-small-cell-lung-cancer
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I think AstraZeneca already knows Durvalumab would work out best with PS Targeting.... instead of AZ's Gefitinib
Lets go Peregrine.....what does the next little round of news include
"Bavituximab is a first-in-class phosphatidylserine (PS)-targeting monoclonal antibody that is the cornerstone of a broad clinical
pipeline." -- Big Pharmas nightmare... unless they are fortunate enough to have The Bavi Edge!
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