Solanezumab binds soluble a-beta. According to the "sink hypothesis" removing peripheral, soluble a-beta should impact the insoluble a-beta deposits in brain plaques, in order to maintain equilibrium between the two forms.
Thanks, Idit. So, is it fair to say that the “sink hypothesis” has been discredited by the latest Solanezumab findings?
“The efficient-market hypothesis may be the foremost piece of B.S. ever promulgated in any area of human knowledge!”
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