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Re: Amatuer17 post# 68589

Tuesday, 07/19/2016 10:10:35 AM

Tuesday, July 19, 2016 10:10:35 AM

Post# of 462738
From the article...

"Despite its importance, the fundamental mechanism of how protein fibrils can self-replicate without any additional machinery is not well understood. In a study published today in the journal Nature Physics, a team led by researchers from the Department of Chemistry at the University of Cambridge used a powerful combination of computer simulations and laboratory experiments to identify the necessary requirements for the self-replication of protein fibrils."

So, are the proteins self-replicating or might they continue to produce because of growing stressed cells via neuroinflamation? If the decline in cell health is exacerbated with age, then it might appear the amyloid plaque is replicating, when in fact it is being produced by more dying cells, which are unable to slough off or prevent the misfolded proteins. The MoA of 2-73 is maintaining cell health.
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