FTM,
"prostaglandin E2 (PGE2) gets in your blood and travels to the hypothalamus in your
brain which controls your body temperature".
Most infections caused by cuts, burns, tooth infections, bug bites are contained within the bodies lymphatic system. I assuming prostaglandin would stay local as well. That's good info, if we're talking about bacterial infections, but were not.
As I'm sure you are aware, there are other factors, or triggers to fever. These include, immune response to viruses, and drug withdrawal. Certain cancers, such as lymphomas can cause fever, especially as an early immune response.
Cancers, and viruses do proliferate inside the body, by hiding from the immune system, and turning B & T cells blind. What I'm trying to figure out is, how Bavituximab can illicit, or trigger an appropriate immune response, reducing the pathogen's survival and proliferating tumor vascular, without causing traditional immune symptomatic responses. This all assumes Bavituximab has an un-blinding effect on cancer specific antigens.
Is the answer to that question in yours and Thurly's rebuttal? I'm not sure but at least it's up for conversation.
I appreciate your opinion, and DD. The same for Thurly.
Regards,
News 
Market Data 
Discover 
AI
