If I'm not mistaken, I think this was actually addressed on the last conference call. It's nice to know that it wasn't due to virological failure. I know the CCR5 tropism can morph to a different subtype of the virus. I wonder if the fact that PRO 140 has the ability to achieve full virological suppression that it can actually slow or stop the morphisms altogether. Maybe this is one of their aces in the hole.
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