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Monday, November 19, 2012 4:58:31 PM
I'm suggesting that over time, Bavituximab might alter (as part of the immune response) and make recognizable (exposed Cancerous Cells) for Germline DNA (new cells), to "NOT" replicate the altered DNA in damaged (cancerous) cells, and eventually arrest cancer proliferation.
Is this not the process of a healthy innate immune system? If not then carcinogen exposure would be catastrophic for the human species and damaged DNA would go unchecked, causing cancer in a much grander scale.
Now if you want an answer, or supporting facts to my theory, then re-read my first post today. If Bavituximab is working then we would be seeing mitochondrial expression, and as a result there would be chemical energy released by (AT), making my theory a possibility.
In Peregrines last PR, they said that they were seeing signalling chemicals, one of which is Nitric Oxide, which I told the board it was, and I'm right. Now I'm saying this is the other Bavituximab proof of action (chemical marker), and if it is then everything I said is possible. That's it, and nothing more.
Lastly, ask yourself this FTM, if Bavituximab is administered and the goal is to become cancer free, and you are in remission, and it never comes back, because of an immune response, what just happened? and everything I've said is possible.
Above is all my opinion,
John
Disclaimer: Every post, and all my views are only speculative. Do not invest money or any other resources based on these post or opinions. Best of luck and do your own due diligence!
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