Avid Bioservices Inc (CDMO)

[goodJohnhunting]

How can you say that Bavituximab is iliciting an immune response, and through this response, and over an extended period of time arresting tumor vascular, and cell proliferation without the presence of (AT) via mitochondria expression, and not, (over time) repair the DNA, thus recognizing innately the tumor it just arrested? Why are you limiting Bavituximab, and not asking yourself, how, and where is this possible?

I'm suggesting that over time, Bavituximab might alter (as part of the immune response) and make recognizable (exposed Cancerous Cells) for Germline DNA (new cells), to "NOT" replicate the altered DNA in damaged (cancerous) cells, and eventually arrest cancer proliferation.

Is this not the process of a healthy innate immune system? If not then carcinogen exposure would be catastrophic for the human species and damaged DNA would go unchecked, causing cancer in a much grander scale.

Now if you want an answer, or supporting facts to my theory, then re-read my first post today. If Bavituximab is working then we would be seeing mitochondrial expression, and as a result there would be chemical energy released by (AT), making my theory a possibility.

In Peregrines last PR, they said that they were seeing signalling chemicals, one of which is Nitric Oxide, which I told the board it was, and I'm right. Now I'm saying this is the other Bavituximab proof of action (chemical marker), and if it is then everything I said is possible. That's it, and nothing more.

Lastly, ask yourself this FTM, if Bavituximab is administered and the goal is to become cancer free, and you are in remission, and it never comes back, because of an immune response, what just happened? and everything I've said is possible.

Above is all my opinion,
John