bapineuzumab does not only bind to soluble abeta in the blood.
it binds directly to abeta on the cell surface. it binds plaques, fibrils, oligimers, dimers and monomers. and as you say, it binds to soluble vascular abeta.
it is an abeta hammer. If it doesn't work, i don't think any of the other amyloiud drugs will.
they only concern i ahve about its clinical effectiveness is that they are treating to late and too short of a duration.
I think they should be treating MCI or prodromal patients and treating them for up to 4 years.
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