So are you saying that even though Ariad's 113 could be more potent against ALK, the cancer develops resistance through other pathways that '113 doesn't inhibit?
That’s exactly my point. A second-line ALK inhibitor may not get the job done if a patient’s resistance to Crizotinib comes from activation of other oncogenes, which is likely to be the case in many instances.
“The efficient-market hypothesis may be the foremost piece of B.S. ever promulgated in any area of human knowledge!”
Register for free to join our community of investors and share your ideas. You will also get access to streaming quotes, interactive charts, trades, portfolio, live options flow and more tools.