ALthough I am not a virologist, I had a chance to look it up for you. The new Viral load is measured at site of replication - skin cells....
I am pasting a link and a paragraph from the link....
"RECURRENT HERPETIC INFECTION (Herpes labialis)
Excitants such as: GI upsets, stress, menses, solar radiation, extreme cold or other infections, will reactivate the virus in around 40% of carriers. This reactivation induces migration from the ganglion to the peripheral epithelial cells where the virus replicate. This new viral load will produce recurrent lesions which are generally less severe than the primary ones. The recurrent lesions on the lips go through several clinical stages which are: burning sensation, erythema of the affected area, vesiculation, ulceration and crust formation. Recurrent herpes simplex, also known as recurrent herpes labialis (cold sores), as the name implies, develops on the lips, generally at the junction of the vermilion and the skin. It begins as a burning sensation which last several hours to one day. Erythema is the second stage which is followed by small coalescing vesicles, which are short lived.
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