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Re: sunspotter post# 371999

Thursday, 08/26/2021 11:12:55 AM

Thursday, August 26, 2021 11:12:55 AM

Post# of 403039
Brilacidin's mechanism is three pronged unlike Remdesivir.

As Farrell and KMBJN succintly discussed at length here recently, the mechanism by which Brilacidin disrupts the viral membrane is unique and is extra-cellular, whereas Remdesivir is intra-cellular which itself presents a problem. Moreover, Brilacidin is anti-inflammatory and anti-bacterial, something that Remdesivir lacks.

A high SI of Remdesivir did enable it to succeed in-vivo and receive an EUA (though the extent of its success is under debate). Isn't there a higher probability for Brilacidin to succeed?

Note: I am not in the bio-medical field, so I used layman's terms to express my understanding. But the gentlemen I referred to above have explained it beautifully elsewhere.
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