That's right. A single molecule of any kind can make an astonishing difference in biological systems. Nevertheless, I think anatabine citrate and MYMD-1 are significantly related to one another, and it seems that the Johns Hopkins researchers agree, as they seem to be testing MYMD-1 for some of the same things that got them working on Anatabloc (anatabine citrate) originally. As I understand it, the work at Johns Hopkins is related to the inhibition of excess inflammation. Inhibition of NF-kB's transcriptional function (related to DNA expression) is the common thread.
Corrections welcome.
Incidentally, I have been told that the difference between two mutually fatal (on transfusion) blood types is a single sugar molecule. I've never confirmed, but it makes sense.