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Sunday, 12/06/2020 8:39:35 AM

Sunday, December 06, 2020 8:39:35 AM

Post# of 44690
Further investigation demonstrated increased intracellular cAMP concentration after VIP administration, and showed that the protective effect of VIP on concanavalin A-induced hepatitis was mediated mainly through VIP receptor 1 (VPAC(1)). These results suggest that VIP is capable of attenuating immune-mediated liver injury in vivo. This effect is associated with its downregulation of critical inflammatory mediators and its upregulation of anti-inflammatory cytokine through VPAC(1), possibly via the cAMP-dependent pathway.

https://pubmed.ncbi.nlm.nih.gov/19222997/
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