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Wednesday, December 27, 2017 8:20:27 PM
"The most well-known tumor-suppressing gene is p53, which gets switched on immediately after DNA is damaged, say, from a sunburn. The protein made from this gene helps cells pause division and repair the damage. Cells with a mutated p53 gene do not pause when their DNA is damaged. Instead, they continue dividing and accumulate more damage as a result. This is why p53 is one of the most commonly mutated genes in cancer."
From this article: https://www.mskcc.org/blog/when-loss-gain-new-tumor-suppressor-gene-identified-follicular-lymphoma
Those who minimize todays PR are either ignorant or disengenuous IMO. The application possibilities for Kevetrin are extremely broad even if only as an adjunct. This phase2a was important to solidifying K's value.
Studying the myc gene and p21 along with p53 will give a larger view of cell processes in regard to cell repair and aptosis.
Another substantive step for IPIX.
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