However he did say: "The inflammation drugs, we could get lucky because we know exactly what we need to do, we need to stop this gene, CD33. So I think that’s maybe 10 to 15 years out."
Maybe he is thinking if you stop plaque, you won't get tangles and if they don't exist or are reduced, you won't get inflammation? If so, why would you develop an inflammation drug?
Certainly prevention is very important---but what happens with the patients who already have plaques and tangles already?
Obviously 'inflammation' is becomming more and more recognized and an important issue with autoimmune and related diseases.
